Question:
And, indeed autopsies of young men who have died in combat leads us to believe the there is some degree of CAD in *everyone* with high LDL and low HDL.
At my last checkup and review of my stress-echo my cardiologist stated that plaque build up on the order of up to 40 and 50% of the arterial cross section are quite common and come and go over time. If this is an accurate observation, then perhaps the military autopsies merely showed instances of the ebb and flow rather than the foundations for a continuous build up over a lifetime.
Response:
The problem is that short of a autopsy we do not have a good way of definitively saying that someone has *no CAD*. And, indeed autopsies of young men who have died in combat leads us to believe the there is some degree of CAD in *everyone* with high LDL and low HDL.
As is *some* degree of CAD in *everyone* with high HDL and low LDL…. Mirek
Response:
The problem is that short of a autopsy we do not have a good way of definitively saying that someone has *no CAD*. And, indeed autopsies of young men who have died in combat leads us to believe the there is some degree of CAD in *everyone* with high LDL and low HDL. As is *some* degree of CAD in *everyone* with high HDL and low LDL….
The latter folks have no CAD on autopsy. Mirek
You remain in my prayers, dear Mirek whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
And, indeed autopsies of young men who have died in combat leads us to believe the there is some degree of CAD in *everyone* with high LDL and low HDL. At my last checkup and review of my stress-echo my cardiologist stated that plaque build up on the order of up to 40 and 50% of the arterial cross section are quite common and come and go over time.
I might believe such a conversation taking place after an angiogram but not after a stress-echo which is not a test that can quantify the degree of plaque build-up. If this is an accurate observation,
It is not. then perhaps the military autopsies merely showed instances of the ebb and flow rather than the foundations for a continuous build up over a lifetime.
That would not explain the pristinely clean coronary arteries of those young men with low LDL and high HDL. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
- Hide quoted text — Show quoted text – The truth of the matter may have little to do with what is currently widely accepted. Science is not a democratic process, and truth is not a matter for a vote. Correct. I find the arguments against the established ideas more than credible. That does not mean these arguments represent the truth. Nor does it mean they are not.
However, I discern that thay do not represent the truth. But I am qualified to judge their merits, especially when it comes to the mathematical sleight of hand employed in interpreting statistical results.
I would suggest you look at the data. You can start with Framingham. The financial interests which back the status quo are formidable, however. The financial interests actually back *new* drugs and *new* targets. Patents do expire afterall. Let’s then hope that we eventually get new drugs that do something helpful, rather than a nightmare like statins and predecessors.
Most folks on statins are not having nightmares with it. Many who argue against them seem to have few vested interests and do support their claims with extensive references in the peer-reviewed literature. All is not what they may seem. Perhaps, but I often run down the references and look at raw data myself. I also focus on papers or books which do provide extensive references to the peer-reviewed literature.
Then you should conclude that LDL should be lowered and HDL should be raised if the goal is to prevent atherosclerosis. Typically these are retirees since they would otherwise have suffered professionally to go public sooner. That is one way to look at it. Another way would be that these folks were always on the fringe and retired early because of this. Irrelevant. Does not change the data, nor my personal results.
The same could be said for your current view. You might want to do some reading on the history of the whole cholesterol/sat fat/heart disease idea. Ravnskov’s book is a quick and thorough read on the subject. I also enjoyed books by Enig, McGee, and a couple of others listed on that site. They all have a hard time explaining the animal models of atherosclerosis. Not at all. The subject is well treated in the literature I’ve been exposed to.
It seems by your next statement, your exposure has been "kill-filtered." And of course animal models are not in general directly relevant or predictive for humans. Until results with animals are actually shown to be reproducible in humans, it cannot be assumed that they will.
The disease that is reproduced in the animal models occurs under conditions that humans are currently exposed to in the societies with high rates of atherosclerotic disease. – Hide quoted text — Show quoted text – One other good cross reference on this is to look at recent books by aging researchers (e.g. Austad, Finch, Kirkwood) as well as studying some nutritional biochemistry (e.g. Stipanuk). Seems a great deal can be found in accordance with the idea that the lipid theory is wrong, and that we should be more worried about refined carbohydrates, processed foods, and chronic imflammatory processes. If you also do some Pubmed searches on research into atherogenesis, you will find a great deal going on in areas unrelated to cholesterol or fat intake. Most regard elevated serum cholesterol as a permissive condition. As distinct from a causative factor. It is irrational to attempt to defeat a normal and essential biochemical condition simply because it is permissive of a potential disease process with a separate cause, especially when the methods are exceedingly crude and on the whole ineffective, and in fact promote a wide range of other disease processes. Studies show that cholesterol lowering is a bad idea for almost everyone.
You are welcome to cite the studies. I would be more than happy to give you my professional comments on each. Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport." Just another questionable way to attack symptoms when instead the cause should be prevented.
This is a observed phenomenon rather than a treatment. Personally I’ve adopted a restricted carbohydrate, high sat-fat diet over a year a go. (I eat mainly whole unprocessed foods, no fast food, and cook everything fresh or eat it raw.) Uh-oh. Not! Best decision I ever made relative to current quality of life improvement. Try not to be so confined to old-school and cardiocentric thinking.
The mission here is to prevent catastrophic cardiovascular events. I do note that since you cannot get past the rather primitive superstitions your were taught as a child, you may well have similar trouble accepting scientific data that conflict with your current medical indoctrination.
Sorry my being openly Christian distracts you. I’ve effortlessly lost all of the excess weight I gained over 25 years, and have had complete relief from several long term health problems, one of them chronically debilitating. I would be concerned about hyperketonemia being behind your loss of appetite. Not even a remote possibility. I have not needed to achieve ketosis for quite a long long time (if I ever did). My appetite is great, but non carbohydrate rich foods do not cause me to gain weight.
They would if you ate more of them. You ought to bone up on how these diets are done and what they achieve past the initial rapid weight loss phases.
It is doubtful that my reviewing your diet methodologies would reveal another explanation for your loss of appetite. Now if you want to argue that my exceptionally stable and low blood sugar levels (achieved via almost constant gluconeogenesis) are harmful then show me the research.
Why would I want to do that? My concern is your possibly having hyperketonemia because of inadequate dietary carbohydrates. Your body stores carbohydrates in the form of glycogen because carbohydrates are an essential macronutrient. Note that high blood sugar levels have much better correlation with cardiovascular disease that any level of any cholesterol fraction.
It seems you are confusing diabetes with high blood sugar. My blood sugar stays locked in at the levels my liver maintains, my triglycerides are very low, and my LDL has remained the same as it was on the low fat diet I was on for 20 years. With the weight loss, it should be lower. It’s not.
Which is why I would be concerned. But my levels have never been very high, and they have followed a pretty typical age-related curve.
Such age-related curves are better explained as a weight-related curve. Other measures have improved, and I’ve had an echocardiogram which showed no sign of disease. An echocardiogram won’t until *after* your heart has suffered injury. Duh!
There can be severe atherosclerotic disease *before* there is injury. President Clinton would be a timely illustrative case in point. He was also low-carbing. Obviously, I’ve suffered no injury to date.
Presumably by not obviously to me. Until I see at least some inkling that I have, I see no reason to alter my course.
That was apparently President Clinton’s philosophy on this topic. For now this is the choice I am making, though I’m keeping my eye on the scientific literature as new findings come out, and of course watching the impact on my own health. So far ignoring the established advice has been all good for me. May that not be your epitaph. Time will tell, and for now I place my bets on nutritional biochemistry and other science over cardiology, as the latter has been a complete and utter failure for all those in my extended family that have had such involvement. None of the drugs, surgeries, or devices has done anything to extend or improve quality of life.
The lifestyle changes and medications work better when done *before* there is a cardiovascular "event." I will try a different path until new data comes out that can change my mind.
Your informed choice for your health and your soul. Alan
You remain in my prayers, dear Alan whom I love. May Christ heal you as only He can. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
The truth of the matter may have little to do with what is currently widely accepted. Science is not a democratic process, and truth is not a matter for a vote. Correct. I find the arguments against the established ideas more than credible. That does not mean these arguments represent the truth.
Nor does it mean they are not. But I am qualified to judge their merits, especially when it comes to the mathematical sleight of hand employed in interpreting statistical results. The financial interests which back the status quo are formidable, however. The financial interests actually back *new* drugs and *new* targets. Patents do expire afterall.
Let’s then hope that we eventually get new drugs that do something helpful, rather than a nightmare like statins and predecessors. Many who argue against them seem to have few vested interests and do support their claims with extensive references in the peer-reviewed literature. All is not what they may seem.
Perhaps, but I often run down the references and look at raw data myself. I also focus on papers or books which do provide extensive references to the peer-reviewed literature. Typically these are retirees since they would otherwise have suffered professionally to go public sooner. That is one way to look at it. Another way would be that these folks were always on the fringe and retired early because of this.
Irrelevant. Does not change the data, nor my personal results. You might want to do some reading on the history of the whole cholesterol/sat fat/heart disease idea. Ravnskov’s book is a quick and thorough read on the subject. I also enjoyed books by Enig, McGee, and a couple of others listed on that site. They all have a hard time explaining the animal models of atherosclerosis.
Not at all. The subject is well treated in the literature I’ve been exposed to. And of course animal models are not in general directly relevant or predictive for humans. Until results with animals are actually shown to be reproducible in humans, it cannot be assumed that they will. One other good cross reference on this is to look at recent books by aging researchers (e.g. Austad, Finch, Kirkwood) as well as studying some nutritional biochemistry (e.g. Stipanuk). Seems a great deal can be found in accordance with the idea that the lipid theory is wrong, and that we should be more worried about refined carbohydrates, processed foods, and chronic imflammatory processes. If you also do some Pubmed searches on research into atherogenesis, you will find a great deal going on in areas unrelated to cholesterol or fat intake. Most regard elevated serum cholesterol as a permissive condition.
As distinct from a causative factor. It is irrational to attempt to defeat a normal and essential biochemical condition simply because it is permissive of a potential disease process with a separate cause, especially when the methods are exceedingly crude and on the whole ineffective, and in fact promote a wide range of other disease processes. Studies show that cholesterol lowering is a bad idea for almost everyone. Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport."
Just another questionable way to attack symptoms when instead the cause should be prevented. Personally I’ve adopted a restricted carbohydrate, high sat-fat diet over a year a go. (I eat mainly whole unprocessed foods, no fast food, and cook everything fresh or eat it raw.) Uh-oh.
Not! Best decision I ever made relative to current quality of life improvement. Try not to be so confined to old-school and cardiocentric thinking. I do note that since you cannot get past the rather primitive superstitions your were taught as a child, you may well have similar trouble accepting scientific data that conflict with your current medical indoctrination. I’ve effortlessly lost all of the excess weight I gained over 25 years, and have had complete relief from several long term health problems, one of them chronically debilitating. I would be concerned about hyperketonemia being behind your loss of
appetite. Not even a remote possibility. I have not needed to achieve ketosis for quite a long long time (if I ever did). My appetite is great, but non carbohydrate rich foods do not cause me to gain weight. You ought to bone up on how these diets are done and what they achieve past the initial rapid weight loss phases. Now if you want to argue that my exceptionally stable and low blood sugar levels (achieved via almost constant gluconeogenesis) are harmful then show me the research. Note that high blood sugar levels have much better correlation with cardiovascular disease that any level of any cholesterol fraction. My blood sugar stays locked in at the levels my liver maintains, my triglycerides are very low, and my LDL has remained the same as it was on the low fat diet I was on for 20 years. With the weight loss, it should be lower.
It’s not. But my levels have never been very high, and they have followed a pretty typical age-related curve. Other measures have improved, and I’ve had an echocardiogram which showed no sign of disease. An echocardiogram won’t until *after* your heart has suffered injury.
Duh! Obviously, I’ve suffered no injury to date. Until I see at least some inkling that I have, I see no reason to alter my course. For now this is the choice I am making, though I’m keeping my eye on the scientific literature as new findings come out, and of course watching the impact on my own health. So far ignoring the established advice has been all good for me. May that not be your epitaph.
Time will tell, and for now I place my bets on nutritional biochemistry and other science over cardiology, as the latter has been a complete and utter failure for all those in my extended family that have had such involvement. None of the drugs, surgeries, or devices has done anything to extend or improve quality of life. I will try a different path until new data comes out that can change my mind. Alan
Response:
Big Macs and fries are *not* low-carb. Clinton was low-carbing maybe 3 months before the discovery of his heart problems, which were caused by a lifetime of junk food, not by his more recent (moderately) low-carb diet.
Response:
Big Macs and fries are *not* low-carb. Clinton was low-carbing maybe 3 months before the discovery of his heart problems, which were caused by a lifetime of junk food, not by his more recent (moderately) low-carb diet.
Step aside, flyweight. It’s about science which we both know is far over your pretty little head. Bob
Response:
There can be severe atherosclerotic disease *before* there is injury. President Clinton would be a timely illustrative case in point. He was also low-carbing. Big Macs and fries are *not* low-carb.
Correct. Clinton was low-carbing maybe 3 months before the discovery of his heart problems, which were caused by a lifetime of junk food, not by his more recent (moderately) low-carb diet.
There is such an entity as accelerated coronary atherosclerosis (CAD). We can induce laboratory animals to go from *no* CAD to *severe* CAD in a matter of 3-6 months. We do this with a high lipid (low carb and low protein) diet. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
– Hide quoted text — Show quoted text – Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport." BTW, as you probably really mean the role of HDL and TC/HDL ratio, how do you think they lost the bet? http://makeashorterlink.com/?L2A913059
Sorry, Andrew, but I do not understand what exactly do you mean. This link is about drug, not about lipid theory. This drug is able to raise HDL and it is also able to remove plague. Did you want to say that this drug demonstrates that low HDL causes CAD? Or what? Mirek
Response:
- Hide quoted text — Show quoted text – Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport." BTW, as you probably really mean the role of HDL and TC/HDL ratio, how do you think they lost the bet? http://makeashorterlink.com/?L2A913059 Sorry, Andrew, but I do not understand what exactly do you mean. This link is about drug, not about lipid theory.
The existence of such a "drug" and its properties supports the theory of "reverse cholesterol transport." This drug is able to raise HDL and it is also able to remove plague. Did you want to say that this drug demonstrates that low HDL causes CAD?
No. Or what?
See above. Mirek
You remain in my prayers, dear Mirek whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
Sorry, Andrew, but I do not understand what exactly do you mean. This link is about drug, not about lipid theory. The existence of such a "drug" and its properties supports the theory of "reverse cholesterol transport."
OK, so we have reverse cholesterol transport. Good to know, but one would expect it based on Framingham study. How does it makes scientists doubting significance of TC levels on heart disease wrong? I think it equivalently makes them right. Mirek
Response:
Sorry, Andrew, but I do not understand what exactly do you mean. This link is about drug, not about lipid theory. The existence of such a "drug" and its properties supports the theory of "reverse cholesterol transport." OK, so we have reverse cholesterol transport. Good to know, but one would expect it based on Framingham study.
The Framingham Heart Study was an epidemiological study. Such studies rarely if ever shed light on the underlying mechanism of an observed phenomenon. How does it makes scientists doubting significance of TC levels on heart disease wrong?
Those opposed to the "cholesterol theory of vascular disease" have generally proposed mechanisms that are independent of cholesterol. "Reverse cholesterol transport" is a mechanism of vascular disease reversal that firmly entrenches the role of cholesterol in the pathophysiology of this disease. I think it equivalently makes them right.
You are entitled to hold unfounded opinions. Mirek
You remain in my prayers, dear Mirek whom I love. May God bless you today. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
How does it makes scientists doubting significance of TC levels on heart disease wrong? Those opposed to the "cholesterol theory of vascular disease" have generally proposed mechanisms that are independent of cholesterol. "Reverse cholesterol transport" is a mechanism of vascular disease reversal that firmly entrenches the role of cholesterol in the pathophysiology of this disease.
Good and reasonable answer. Anyway, just because drug that is _supposed_ to perform reverse cholesterol transport decreases plague does not neccessary imply that LDL levels alone are causative to creation of this plague. Permisive condition – perhaps. But as long as there are people with high LDL, low HDL and no CAD, things seem to be more complicated than simple cholesterol balance. Mirek
Response:
- Hide quoted text — Show quoted text – How does it makes scientists doubting significance of TC levels on heart disease wrong? Those opposed to the "cholesterol theory of vascular disease" have generally proposed mechanisms that are independent of cholesterol. "Reverse cholesterol transport" is a mechanism of vascular disease reversal that firmly entrenches the role of cholesterol in the pathophysiology of this disease. Good and reasonable answer. Anyway, just because drug that is _supposed_ to perform reverse cholesterol transport decreases plague does not neccessary imply that LDL levels alone are causative to creation of this plague.
Those who subscribe to the "cholesterol theory of vascular disease" are not claiming that LDL levels *alone* cause the disease but rather that it is an important risk factor that should be lowered if one of the goals is prevention (either primary or secondary). Permisive condition – perhaps. But as long as there are people with high LDL, low HDL and no CAD, things seem to be more complicated than simple cholesterol balance.
The problem is that short of a autopsy we do not have a good way of definitively saying that someone has *no CAD*. And, indeed autopsies of young men who have died in combat leads us to believe the there is some degree of CAD in *everyone* with high LDL and low HDL. Mirek
You remain in my prayers, dear Mirek whom I love. May God bless you on this Lord’s day. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport."
What do you exactly mean by "reverse cholesterol transport"? HDL and atherogenic ratio? (Please, I do not expect detailed answer, but some link would be helpful.) Mirek
Response:
Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport."
BTW, as you probably really mean the role of HDL and TC/HDL ratio, how do you think they lost the bet? Mirek
Response:
Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport." BTW, as you probably really mean the role of HDL and TC/HDL ratio, how do you think they lost the bet?
http://makeashorterlink.com/?L2A913059 Mirek
YOu remain in my prayers, dear Mirek whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking). These folks have lost the bet with the discovery of "reverse cholesterol transport." What do you exactly mean by "reverse cholesterol transport"?
http://makeashorterlink.com/?L2A913059 HDL and atherogenic ratio?
Not quite. (Please, I do not expect detailed answer, but some link would be helpful.)
(You’ve got it) Mirek
You remain in my prayers, dear Mirek whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
What do you exactly mean by "reverse cholesterol transport"? http://makeashorterlink.com/?L2A913059
I gess it is not the most informative link, but interesting anyway. I just hope these drugs will be available when I will need them:) Mirek
Response:
- Hide quoted text — Show quoted text – Actauly, in you case, any dieting in long term would likely end in gaining a weight. Not for the 2PD Approach. How many people are on 2PD for more than 5 years? More than a busy cardiologist like me can track 
That’s a pretty good trick, since you state on your website that you were watching the film that inspired it in 1998.
<LOL Bob
Response:
Actauly, in you case, any dieting in long term would likely end in gaining a weight. Not for the 2PD Approach. How many people are on 2PD for more than 5 years? More than a busy cardiologist like me can track
Translation: None. HTH Bob
Response:
Actauly, in you case, any dieting in long term would likely end in gaining a weight. Not for the 2PD Approach. How many people are on 2PD for more than 5 years? More than a busy cardiologist like me can track
That’s a pretty good trick, since you state on your website that you were watching the film that inspired it in 1998. — "There’s a seeker born every minute."
Response:
The truth of the matter may have little to do with what is currently widely accepted. Science is not a democratic process, and truth is not a matter for a vote.
Correct. I find the arguments against the established ideas more than credible.
That does not mean these arguments represent the truth. The financial interests which back the status quo are formidable, however.
The financial interests actually back *new* drugs and *new* targets. Patents do expire afterall. Many who argue against them seem to have few vested interests and do support their claims with extensive references in the peer-reviewed literature.
All is not what they may seem. Typically these are retirees since they would otherwise have suffered professionally to go public sooner.
That is one way to look at it. Another way would be that these folks were always on the fringe and retired early because of this. You might want to do some reading on the history of the whole cholesterol/sat fat/heart disease idea. Ravnskov’s book is a quick and thorough read on the subject. I also enjoyed books by Enig, McGee, and a couple of others listed on that site.
They all have a hard time explaining the animal models of atherosclerosis. One other good cross reference on this is to look at recent books by aging researchers (e.g. Austad, Finch, Kirkwood) as well as studying some nutritional biochemistry (e.g. Stipanuk). Seems a great deal can be found in accordance with the idea that the lipid theory is wrong, and that we should be more worried about refined carbohydrates, processed foods, and chronic imflammatory processes. If you also do some Pubmed searches on research into atherogenesis, you will find a great deal going on in areas unrelated to cholesterol or fat intake.
Most regard elevated serum cholesterol as a permissive condition. Seems quite a few researchers are betting that answers to the CHD puzzle will be found elsewhere (and implicitly that current hypotheses are lacking).
These folks have lost the bet with the discovery of "reverse cholesterol transport." Personally I’ve adopted a restricted carbohydrate, high sat-fat diet over a year a go. (I eat mainly whole unprocessed foods, no fast food, and cook everything fresh or eat it raw.)
Uh-oh. I’ve effortlessly lost all of the excess weight I gained over 25 years, and have had complete relief from several long term health problems, one of them chronically debilitating.
I would be concerned about hyperketonemia being behind your loss of appetite. My blood sugar stays locked in at the levels my liver maintains, my triglycerides are very low, and my LDL has remained the same as it was on the low fat diet I was on for 20 years.
With the weight loss, it should be lower. Other measures have improved, and I’ve had an echocardiogram which showed no sign of disease.
An echocardiogram won’t until *after* your heart has suffered injury. For now this is the choice I am making, though I’m keeping my eye on the scientific literature as new findings come out, and of course watching the impact on my own health. So far ignoring the established advice has been all good for me.
May that not be your epitaph. Alan
You will be in my prayers, dear neighbor whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
Actauly, in you case, any dieting in long term would likely end in gaining a weight. Not for the 2PD Approach. How many people are on 2PD for more than 5 years?
More than a busy cardiologist like me can track You remain in my prayers, dear neighbor whom I love. Servant to the humblest person in the universe, Andrew — Dr. Andrew B. Chung, MD/PhD Board-Certified Cardiologist http://www.heartmdphd.com/ ** Who is the humblest person in the universe? http://makeashorterlink.com/?L26062048 What is all this about? http://makeashorterlink.com/?R20632B48 Is this spam? http://makeashorterlink.com/?N69721867
Response:
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Fortunately, green tea contains little 